Comments on: Milk consumption tied to Parkinson’s disease http://bacteriality.com/2009/04/04/milk/ Tue, 29 Jun 2010 15:10:28 +0000 hourly 1 http://wordpress.org/?v=3.0 By: Amy Proal http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-18494 Amy Proal Sat, 14 Nov 2009 02:11:48 +0000 http://bacteriality.com/?p=746#comment-18494 Crispy, At the risk of sounding rude and in an effort to maintain clarity, I don't support your conclusions. Best, Amy Crispy,

At the risk of sounding rude and in an effort to maintain clarity, I don’t support your conclusions.

Best,
Amy

]]> By: crispy http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-18491 crispy Sat, 14 Nov 2009 01:04:12 +0000 http://bacteriality.com/?p=746#comment-18491 here is a quote from the article (New Scientist): "Vitamin E is not an antioxidant. In fact it must be protected against oxidation," says Angelo Azzi, a biochemist at Tufts University in Boston, Massachusetts." what kind of a biochemist is he? Vit E is antioxidant BECAUSE it neutralizes (captures, as some put it) free radicals; in that process it becomes oxidized, the free radical being reduced. To remain active indeed it has to be protected against oxidation (from O2 for ex). Vit E, acting as an antioxidant, will prevent oil from becoming rancid (try it if You don't believe me w 2 bottles of say olive oil, 1 w added vit E and the other without exposed to natural heat and sunlight). The large reduction of incidence of cardiovascular disease from Vit E, in some studies, speaks for itself. I don't know why other studies failed to show similar results. Could it be a different amount of E? Or more likely the form of synthetic Vit E given - instead of MIXED TOCOPHEROLS (what I take)? YOU have to judge for yourself and perhaps look at Life Extension magazine with many articles on Vit E and other antioxidants. Yes, LE sells Vit and supplements (Expensive on top of that) and can be seen as having a conflict of interest BUT you MUST look at their Medical advisory board before you jump to conclusions: Md's, professors of medicine etc (not a journalist writing in the New Scientist probably on behalf of pharma interests!). End of debate for me. here is a quote from the article (New Scientist):
“Vitamin E is not an antioxidant. In fact it must be protected against oxidation,” says Angelo Azzi, a biochemist at Tufts University in Boston, Massachusetts.”
what kind of a biochemist is he?
Vit E is antioxidant BECAUSE it neutralizes (captures, as some put it) free radicals; in that process it becomes oxidized, the free radical being reduced. To remain active indeed it has to be protected against oxidation (from O2 for ex).
Vit E, acting as an antioxidant, will prevent oil from becoming rancid (try it if You don’t believe me w 2 bottles of say olive oil, 1 w added vit E and the other without exposed to natural heat and sunlight).
The large reduction of incidence of cardiovascular disease from Vit E, in some studies, speaks for itself. I don’t know why other studies failed to show similar results. Could it be a different amount of E? Or more likely the form of synthetic Vit E given – instead of MIXED TOCOPHEROLS (what I take)?
YOU have to judge for yourself and perhaps look at Life Extension magazine with many articles on Vit E and other antioxidants.
Yes, LE sells Vit and supplements (Expensive on top of that) and can be seen as having a conflict of interest BUT you MUST look at their Medical advisory board before you jump to conclusions:
Md’s, professors of medicine etc (not a journalist writing in the New Scientist probably on behalf of pharma interests!).
End of debate for me.

]]> By: crispy http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-18490 crispy Sat, 14 Nov 2009 00:35:04 +0000 http://bacteriality.com/?p=746#comment-18490 On Antioxidants and incidence of disease (Paul Albert Aug 6 09): the New Scientist article you mention is worth reading by anyone interested in ia well written piece quoting people who know nothing about health and antioxidants: look at there credentials; nothing is hidden. No medical doctor is quoted ; no-one with opposite views with credentials in healthcare is interviewed. Furthermore, if bias needed to be proven, some benefits of antioxidants are acknowledged in one paragraph but the implications seem to be ignored by the author.. Let me give 1 ex of inaccuracy in this article: A big mistake is made by one of the scientist quoted who does not understand the difference between antioxidant capacity and reducing capacity (measured by Redox potential). This is like comparing calcium carbonate (with its known"anti-acid capacity" = buffering capacity) and the lack of high Ph in water + CaCO3 (Ph akin to redox potential). Perhaps it's not the best ex since CaCO3 is not soluble in water but i can't think of abetter one now. Antioxidants act like a buffer against free radicals (capturing them in fact).They do not affect the redox potential in SO whose redox potential is within normal values allowing life. The chemist who gave me the same link said this article proved the free radical theory of aging and disease was invalid... He is wrong and I hope my comments helped him to understand why )he understands chemistry). So much for this article which ignores thousands of publications linking - if not PROVING - the benefits of diets rich in antioxidants and supplementation above and beyond diet. I am not blaming you for quoting this paper: look at it again in detail; you'll see what I said is true and we can quote it as a biased, unscientific look at the subject of antioxidants. RE: Vit C and UA; I assume the reduction of uric acid levels by Vit C (which I haven't verified) is largely compensated by the antioxidant effect of Vit C. In Ph terms I'd assume Vit C is a better/stronger buffer than uric acid or that the loss of buffer from lower UA is largely compensated by increased buffer from Vit C. I hope this doesn't cause further confusion. Acai is widely overrated (not the highest ORAC value!) and is expensive. I'd stick to Kiwi and berries! I decided not to follow MP but the above is unrelated. On Antioxidants and incidence of disease (Paul Albert Aug 6 09): the New Scientist article you mention is worth reading by anyone interested in ia well written piece quoting people who know nothing about health and antioxidants: look at there credentials; nothing is hidden. No medical doctor is quoted ; no-one with opposite views with credentials in healthcare is interviewed. Furthermore, if bias needed to be proven, some benefits of antioxidants are acknowledged in one paragraph but the implications seem to be ignored by the author..
Let me give 1 ex of inaccuracy in this article:
A big mistake is made by one of the scientist quoted who does not understand the difference between antioxidant capacity and reducing capacity (measured by Redox potential).
This is like comparing calcium carbonate (with its known”anti-acid capacity” = buffering capacity) and the lack of high Ph in water + CaCO3 (Ph akin to redox potential).
Perhaps it’s not the best ex since CaCO3 is not soluble in water but i can’t think of abetter one now.
Antioxidants act like a buffer against free radicals (capturing them in fact).They do not affect the redox potential in SO whose redox potential is within normal values allowing life.
The chemist who gave me the same link said this article proved the free radical theory of aging and disease was invalid… He is wrong and I hope my comments helped him to understand why )he understands chemistry).
So much for this article which ignores thousands of publications linking – if not PROVING – the benefits of diets rich in antioxidants and supplementation above and beyond diet.
I am not blaming you for quoting this paper: look at it again in detail; you’ll see what I said is true and we can quote it as a biased, unscientific look at the subject of antioxidants.
RE: Vit C and UA; I assume the reduction of uric acid levels by Vit C (which I haven’t verified) is largely compensated by the antioxidant effect of Vit C. In Ph terms I’d assume Vit C is a better/stronger buffer than uric acid or that the loss of buffer from lower UA is largely compensated by increased buffer from Vit C.
I hope this doesn’t cause further confusion.
Acai is widely overrated (not the highest ORAC value!) and is expensive. I’d stick to Kiwi and berries!
I decided not to follow MP but the above is unrelated.

]]> By: Joseph Campisi http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17973 Joseph Campisi Mon, 24 Aug 2009 17:47:12 +0000 http://bacteriality.com/?p=746#comment-17973 Of further interest, the herb Artemisia, a traditional remedy for infection, is from my readings, probably the most common herb used by the Lyme community to treat comorbid mycoplasma infection. It has scientifically proven to be effective against several mycoplasma species. Also Dr. Virginia Livingston, working here in Newark at Saint Michael’s Hospital in the 1950s eventually developed an autogenous vaccine for a cell wall deficient bacteria. Her work has been updated by Dr. Alan Cantwell, who implicated cell wall deficient mycoplasma species in scleroderma, sarcoidosis, sarcoma, and Hodgkin’s. The widespread plant phenol hydroxytyrosol has demonstrated antimycoplasmal activity, and has been suggested as an ancillary treatment for those with antibiotic resistant mycoplasmas. Livingston VW, Alexander-Jackson E Mycobacterial forms in myocardial vascular disease. J Am Med Womens Assoc. 1965 May;20:449-52. Al-Momani W, Abu-Basha E, Janakat S, Nicholas RA, Ayling RD. (2007). In vitro antimycoplasmal activity of six Jordanian medicinal plants against three Mycoplasma species. Trop Anim Health Prod. 2007 Oct;39(7):515-9. Livingston VW, Alexander-Jackson E (September 1965). "An experimental biologic approach to the treatment of neoplastic disease; determination of actinomycin in urine and cultures as an aid to diagnosis and prognosis". J Am Med Womens Assoc 20 (9): 858–66. Livingston VW, Livingston AM (June 1974). "Some cultural, immunological, and biochemical properties of Progenitor cryptocides". Trans N Y Acad Sci 36 (6): 569–82. Wuerthele-Caspe V, Alexander-Jackson E, Anderson JA, Hillier J, Allen RM, Smith LW (December 1950). "Cultural properties and pathogenicity of certain microorganisms obtained from various proliferative and neoplastic diseases". Am. J. Med. Sci. 220 (6): 638–46. Cantwell AR Jr, Craggs E, Swatek F, Wilson JW. Unusual acid-fast bacteria in panniculitis. Arch Dermatol. 1966 Aug;94(2):161-7. Cantwell AR Jr, Wilson JW. Scleroderma with ulceration secondary to atypical mycobacteria. Arch Dermatol. 1966 Nov;94(5):663-4. Cantwell AR Jr, Craggs E, Wilson JW, Swatek F. Acid-fast bacteria as a possible cause of scleroderma. Dermatologica. 1968;136(3):141-50. Cantwell AR Jr, Kelso DW. Acid-fast bacteria in scleroderma and morphea. Arch Dermatol. 1971 Jul;104(1):21-5. Cantwell AR Jr. Variably acid-fast bacteria in a case of systemic sarcoidosis and hypodermitis sclerodermiformis. Dermatologica. 1981;163(3):239-48. Cantwell AR Jr. Bacteriologic investigation and histologic observations of variably acid-fast bacteria in three cases of cutaneous Kaposi's sarcoma. Growth. 1981 Summer;45(2):79-89. Cantwell AR Jr. Histologic observations of variably acid-fast coccoid forms suggestive of cell wall deficient bacteria in Hodgkin's disease: a report of four cases. Growth. 1981 Autumn;45(3):168-87. Pio Maria Furneri, Anna Piperno, Antonella Sajia, and Giuseppe Bisignano (2004). Antimycoplasmal Activity of Hydroxytyrosol, Antimicrob Agents Chemother. 2004 December; 48(12): 4892–4894. Cowan, M. M. 1999. Plant products as antimicrobial agents. Clin. Microbiol. Rev. 12:564-582. Of further interest, the herb Artemisia, a traditional remedy for infection, is from my readings, probably the most common herb used by the Lyme community to treat comorbid mycoplasma infection. It has scientifically proven to be effective against several mycoplasma species.
Also Dr. Virginia Livingston, working here in Newark at Saint Michael’s Hospital in the 1950s eventually developed an autogenous vaccine for a cell wall deficient bacteria.
Her work has been updated by Dr. Alan Cantwell, who implicated cell wall deficient mycoplasma species in scleroderma, sarcoidosis, sarcoma, and Hodgkin’s.
The widespread plant phenol hydroxytyrosol has demonstrated antimycoplasmal activity, and has been suggested as an ancillary treatment for those with antibiotic resistant mycoplasmas.
Livingston VW, Alexander-Jackson E Mycobacterial forms in myocardial vascular disease. J Am Med Womens Assoc. 1965 May;20:449-52.

Al-Momani W, Abu-Basha E, Janakat S, Nicholas RA, Ayling RD. (2007). In vitro antimycoplasmal activity of six Jordanian medicinal plants against three Mycoplasma species. Trop Anim Health Prod. 2007 Oct;39(7):515-9.

Livingston VW, Alexander-Jackson E (September 1965). “An experimental biologic approach to the treatment of neoplastic disease; determination of actinomycin in urine and cultures as an aid to diagnosis and prognosis”. J Am Med Womens Assoc 20 (9): 858–66.
Livingston VW, Livingston AM (June 1974). “Some cultural, immunological, and biochemical properties of Progenitor cryptocides”. Trans N Y Acad Sci 36 (6): 569–82.

Wuerthele-Caspe V, Alexander-Jackson E, Anderson JA, Hillier J, Allen RM, Smith LW (December 1950). “Cultural properties and pathogenicity of certain microorganisms obtained from various proliferative and neoplastic diseases”. Am. J. Med. Sci. 220 (6): 638–46.

Cantwell AR Jr, Craggs E, Swatek F, Wilson JW. Unusual acid-fast bacteria in panniculitis. Arch Dermatol. 1966 Aug;94(2):161-7.

Cantwell AR Jr, Wilson JW. Scleroderma with ulceration secondary to atypical mycobacteria. Arch Dermatol. 1966 Nov;94(5):663-4.

Cantwell AR Jr, Craggs E, Wilson JW, Swatek F. Acid-fast bacteria as a possible cause of scleroderma. Dermatologica. 1968;136(3):141-50.

Cantwell AR Jr, Kelso DW. Acid-fast bacteria in scleroderma and morphea. Arch Dermatol. 1971 Jul;104(1):21-5.

Cantwell AR Jr. Variably acid-fast bacteria in a case of systemic sarcoidosis and hypodermitis sclerodermiformis. Dermatologica. 1981;163(3):239-48.

Cantwell AR Jr. Bacteriologic investigation and histologic observations of variably acid-fast bacteria in three cases of cutaneous Kaposi’s sarcoma. Growth. 1981 Summer;45(2):79-89.

Cantwell AR Jr. Histologic observations of variably acid-fast coccoid forms suggestive of cell wall deficient bacteria in Hodgkin’s disease: a report of four cases. Growth. 1981 Autumn;45(3):168-87.

Pio Maria Furneri, Anna Piperno, Antonella Sajia, and Giuseppe Bisignano (2004). Antimycoplasmal Activity of Hydroxytyrosol, Antimicrob Agents Chemother. 2004 December; 48(12): 4892–4894.

Cowan, M. M. 1999. Plant products as antimicrobial agents. Clin. Microbiol. Rev. 12:564-582.

]]> By: Joseph Campisi http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17971 Joseph Campisi Sun, 23 Aug 2009 15:47:27 +0000 http://bacteriality.com/?p=746#comment-17971 Hi Paul, My budget health insurance covers all those tests, since they are not exotic but fundamental tests. Due to out of control medical costs, out of pocket for anything is getting more difficult; but that is another discussion topic. The premise for testing for essential vitamins, minerals, and fatty acids, is the same for testing for vitamin D in many ways. They are called vitamins and essential because they are the fundamental parameters of a healthy metabolism. I find it incredulous that few doctors care about the overall health of patients; naturopaths and orthomolecular medical doctors are notable exceptions. My goal is inexpensive practical cures; however, most diseases are much less expensive to prevent. Eating the right foods, and perhaps a few inexpensive supplements, can restore metabolism to a healthy homeostasis, once illuminated by some diagnostic testing. For example, it is much better to take a few tablespoons of coconut oil, eat tomatoes, consume whole dark grapes (skin and seeds), and use knotweed shoots in salad to PREVENT candida (coconut fatty acids, lycopene and resveratrol kill candida), than to try to remove candida once it has penetrated the intestinal epithelium and taken up residence in blood and organs. I am not arguing against the MP at all. First, I am just suggesting for those not already suffering from mycobacterial over growth, there are dietary, herbal, and supplements that have been scientifically researched to have antimycobacterial properties, which can be logically expected to provide an inhospitable internal environment for mycobacteria to get a foothold. Secondly, does MP have a 100% cure rate? Perhaps for those not cured by MP alone, an ancillary enhancement using a combination of MP and aforementioned nutraceuticals would improve the cure rate for those intractable outliers. For example, at a meeting of the American College of Chest Physicians on October 29, 2008 by Gilda Sapphire Erguiza, M.D. of the Philippine Children's Medical Center and Daniel Rauch, M.D. of the New York University Langone Medical Center, their results of a study in treating mycobacterium tuberculosis, a combination of coconut oil and antibiotics was found to be more effective than antibiotics alone. Your point about immune suppressing effects of fish oil is significant. In various protocols for a variety of diseases, at particular stages of disease progression, the use of immune system enhancement can be contraindicated. To intuitively suggest that immune enhancement is invariably the correct course, while intuitively plausible, is not scientifically correct. While fish oil is established to contribute to the prevention of heart disease, cancer, and depression; depending on the protocol to treat a disease chosen, fish oil may, or may not be appropriate as an ancillary treatment. Take care, Joe Hi Paul, My budget health insurance covers all those tests, since they are not exotic but fundamental tests. Due to out of control medical costs, out of pocket for anything is getting more difficult; but that is another discussion topic. The premise for testing for essential vitamins, minerals, and fatty acids, is the same for testing for vitamin D in many ways. They are called vitamins and essential because they are the fundamental parameters of a healthy metabolism. I find it incredulous that few doctors care about the overall health of patients; naturopaths and orthomolecular medical doctors are notable exceptions.
My goal is inexpensive practical cures; however, most diseases are much less expensive to prevent. Eating the right foods, and perhaps a few inexpensive supplements, can restore metabolism to a healthy homeostasis, once illuminated by some diagnostic testing.
For example, it is much better to take a few tablespoons of coconut oil, eat tomatoes, consume whole dark grapes (skin and seeds), and use knotweed shoots in salad to PREVENT candida (coconut fatty acids, lycopene and resveratrol kill candida), than to try to remove candida once it has penetrated the intestinal epithelium and taken up residence in blood and organs.
I am not arguing against the MP at all.
First, I am just suggesting for those not already suffering from mycobacterial over growth, there are dietary, herbal, and supplements that have been scientifically researched to have antimycobacterial properties, which can be logically expected to provide an inhospitable internal environment for mycobacteria to get a foothold.
Secondly, does MP have a 100% cure rate? Perhaps for those not cured by MP alone, an ancillary enhancement using a combination of MP and aforementioned nutraceuticals would improve the cure rate for those intractable outliers.
For example, at a meeting of the American College of Chest Physicians on October 29, 2008 by Gilda Sapphire Erguiza, M.D. of the Philippine Children’s Medical Center and Daniel Rauch, M.D. of the New York University Langone Medical Center, their results of a study in treating mycobacterium tuberculosis, a combination of coconut oil and antibiotics was found to be more effective than antibiotics alone.
Your point about immune suppressing effects of fish oil is significant. In various protocols for a variety of diseases, at particular stages of disease progression, the use of immune system enhancement can be contraindicated. To intuitively suggest that immune enhancement is invariably the correct course, while intuitively plausible, is not scientifically correct. While fish oil is established to contribute to the prevention of heart disease, cancer, and depression; depending on the protocol to treat a disease chosen, fish oil may, or may not be appropriate as an ancillary treatment.
Take care, Joe

]]> By: Paul Albert http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17966 Paul Albert Sun, 23 Aug 2009 05:37:33 +0000 http://bacteriality.com/?p=746#comment-17966 Hi Joseph, Wow, that's a lot of supplements! Maybe some of the supplements lead to a bacterial die-off reaction, but then again something like fish oil has definitely been shown to slow immune activity: http://mpkb.org/doku.php/home:othertreatments:efas I'm sure you would disagree, but, compared to Benicar and the MP antibiotics, we know relatively little about how these supplements affect the immune system. Besides, our problem is not in generating an immune response. Almost every sick patient on the MP has been able to generate a pronounced a die-off reaction. So, we're right now we're not interested in supplements. We're interested in using medications that have clearly defined actions at the molecular level. As for the tests you mention, those tend to be rather expensive and most patients can't afford them. Best, Paul Hi Joseph,

Wow, that’s a lot of supplements! Maybe some of the supplements lead to a bacterial die-off reaction, but then again something like fish oil has definitely been shown to slow immune activity:
http://mpkb.org/doku.php/home:othertreatments:efas

I’m sure you would disagree, but, compared to Benicar and the MP antibiotics, we know relatively little about how these supplements affect the immune system.

Besides, our problem is not in generating an immune response. Almost every sick patient on the MP has been able to generate a pronounced a die-off reaction.

So, we’re right now we’re not interested in supplements. We’re interested in using medications that have clearly defined actions at the molecular level.

As for the tests you mention, those tend to be rather expensive and most patients can’t afford them.

Best,
Paul

]]> By: Joseph Campisi http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17958 Joseph Campisi Sat, 22 Aug 2009 16:51:40 +0000 http://bacteriality.com/?p=746#comment-17958 Amy, in response to your previous comments: From personally reading hundreds of reports on using samento and cumanda to treat Lyme, babesia, and other microbes, a most common comment is that it does indeed exacerbate symptoms purportedly via the Herxheimer reaction of pathogen die off. The elecampane research mentioned concerning MRSA has been conducted by Susan O'Shea, a research student at Cork Institute of Technology (CIT), Ireland. The Cork Institute of Technology evolved from the Royal Cork Institution in 1807 , and the Crawford Municipal Technical Institute which was founded in 1912. It seems like a legitimate institution. Furthermore, elcampane has been demonstrated effective against mycobacteria by Cantrell CL, Abate L, Fronczek FR, Franzblau SG, Quijano L, Fischer NH in "Antimycobacterial eudesmanolides from Inula helenium and Rudbeckia subtomentosa," Planta Medica, 1999 May;65(4):351-5, reporting that "elcampane root extracts exhibited significant activity against Mycobacterium tuberculosis." A National Library of Medicine "Pubmed" search on antimycobacterial plants returns over one hundred research journal articles. Antimycobacterial phytochemical agents have a considerable research literature. There are many effective natural cures. In the USA physicians face malpractice lawsuits if their protocol is not FDA approved. An FDA approval , and the necessary machinations to bring a new drug to market, is currently estimated to cost one Billion dollars. Who is going to finance studies for all the natural cures that are inexpensive and non-patentable, and therefore, not profitable? USA medical practice is based on profit, not on health. Amy, in response to your previous comments:
From personally reading hundreds of reports on using samento and cumanda to treat Lyme, babesia, and other microbes, a most common comment is that it does indeed exacerbate symptoms purportedly via the Herxheimer reaction of pathogen die off. The elecampane research mentioned concerning MRSA has been conducted by Susan O’Shea, a research student at Cork Institute of Technology (CIT), Ireland.
The Cork Institute of Technology evolved from the Royal Cork Institution in 1807 , and the Crawford Municipal Technical Institute which was founded in 1912. It seems like a legitimate institution. Furthermore, elcampane has been demonstrated effective against mycobacteria by Cantrell CL, Abate L, Fronczek FR, Franzblau SG, Quijano L, Fischer NH in “Antimycobacterial eudesmanolides from Inula helenium and Rudbeckia subtomentosa,” Planta Medica, 1999 May;65(4):351-5, reporting that “elcampane root extracts exhibited significant activity against Mycobacterium tuberculosis.” A National Library of Medicine “Pubmed” search on antimycobacterial plants returns over one hundred research journal articles. Antimycobacterial phytochemical agents have a considerable research literature.
There are many effective natural cures. In the USA physicians face malpractice lawsuits if their protocol is not FDA approved. An FDA approval , and the necessary machinations to bring a new drug to market, is currently estimated to cost one Billion dollars. Who is going to finance studies for all the natural cures that are inexpensive and non-patentable, and therefore, not profitable? USA medical practice is based on profit, not on health.

]]> By: Joseph Campisi http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17957 Joseph Campisi Sat, 22 Aug 2009 16:16:18 +0000 http://bacteriality.com/?p=746#comment-17957 In cases of sarcoidosis, why not consider lab work for: All vitamin and mineral levels? Infectious microbes? Heavy metals, biotoxins, and exotoxins? Food intolerances? Instead of prednisone (or ancillary to) why not consider fish oil, ginger, curcumin, boswellia, rosmarinic acid, and a list of other natural anti-inflammatory agents? In cases of sarcoidosis, why not consider lab work for:
All vitamin and mineral levels?
Infectious microbes?
Heavy metals, biotoxins, and exotoxins?
Food intolerances?
Instead of prednisone (or ancillary to) why not consider fish oil, ginger, curcumin, boswellia, rosmarinic acid, and a list of other natural
anti-inflammatory agents?

]]> By: Claire http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17941 Claire Wed, 19 Aug 2009 00:21:00 +0000 http://bacteriality.com/?p=746#comment-17941 Thank you Amy and Paul for the great article and for your efforts to help raise concern about D. Claire P.S. I certainly hope Daniel takes you up on your suggestion that he read the MP related research, as this comment alone "there are studies that show elevated serum calcium levels, depressed magnesium levels, and depressed vitamin D levels" let me know that he had not. Had he, he may have put forward a more powerful argument--that is, if one exists. Thank you Amy and Paul for the great article and for your efforts to help raise concern about D.

Claire

P.S. I certainly hope Daniel takes you up on your suggestion that he read the MP related research, as this comment alone “there are studies that show elevated serum calcium levels, depressed magnesium levels, and depressed vitamin D levels” let me know that he had not. Had he, he may have put forward a more powerful argument–that is, if one exists.

]]> By: Amy Proal http://bacteriality.com/2009/04/04/milk/comment-page-1/#comment-17918 Amy Proal Wed, 12 Aug 2009 00:54:37 +0000 http://bacteriality.com/?p=746#comment-17918 Hi Daphne, Sorry to hear your have sarcoidosis. I understand how confusing it must be to get a diagnosis of sarcoidosis and hear conflicting views on how to best treat it. I strongly discourage you from taking Prednisone to treat your disease. The following article discusses the negative effects of Prednisone and will give you a more specific idea of why you might not want to take it. http://mpkb.org/doku.php/home:othertreatments:corticosteroids If you opt not to go the Prednisone route, I believe the MP is the choice that makes the most sense. There's strong evidence for bacteria in sarcoidosis and our other patients using the MP to target these bacteria are doing quite well. I'm sorry we can't give your doctor the Phase III data he wants, but we are beginning a clinical trial at West China Hospital, the largest clinical hospital in the world and a center for the Cochrane Collaboration. http://mpkb.org/doku.php/home:arf:072109 In order to better understand our views on vitamin D, take a look at this article. http://mpkb.org/doku.php/home:pathogenesis:vitamind Here's an article on Benicar safety: http://mpkb.org/doku.php/home:protocol:olmesartan:safety You may want to show your doctor our organization's peer-reviewed papers and presentations: http://mpkb.org/doku.php/home:publications Ultimately, the manner in which you treat your sarcoidosis is up to you. If you read enough about the Marshall Protocol and feel it is the best treatment option for your condition and your doctor does not believe it is the right treatment option, you should consider finding another doctor. Here is an article that talks about how to find a doctor who might be supportive. http://mpkb.org/doku.php/home:starting:physician:finding Good luck! Best, Amy Hi Daphne,

Sorry to hear your have sarcoidosis. I understand how confusing it must be to get a diagnosis of sarcoidosis and hear conflicting views on how to best treat it.

I strongly discourage you from taking Prednisone to treat your disease. The following article discusses the negative effects of Prednisone and will give you a more specific idea of why you might not want to take it.
http://mpkb.org/doku.php/home:othertreatments:corticosteroids

If you opt not to go the Prednisone route, I believe the MP is the choice that makes the most sense. There’s strong evidence for bacteria in sarcoidosis and our other patients using the MP to target these bacteria are doing quite well.

I’m sorry we can’t give your doctor the Phase III data he wants, but we are beginning a clinical trial at West China Hospital, the largest clinical hospital in the world and a center for the Cochrane Collaboration.
http://mpkb.org/doku.php/home:arf:072109

In order to better understand our views on vitamin D, take a look at this article.
http://mpkb.org/doku.php/home:pathogenesis:vitamind

Here’s an article on Benicar safety:
http://mpkb.org/doku.php/home:protocol:olmesartan:safety

You may want to show your doctor our organization’s peer-reviewed papers and presentations:
http://mpkb.org/doku.php/home:publications

Ultimately, the manner in which you treat your sarcoidosis is up to you. If you read enough about the Marshall Protocol and feel it is the best treatment option for your condition and your doctor does not believe it is the right treatment option, you should consider finding another doctor.

Here is an article that talks about how to find a doctor who might be supportive.
http://mpkb.org/doku.php/home:starting:physician:finding

Good luck!

Best,
Amy

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