Sick ever since childhood, this resident of New Jersey finally hit rock bottom after developing diabetes and becoming blind in one eye. Not to mention the fact that by the mid 80s his bowels were seriously affected and sarcoidosis had spread throughout much of his body. Yet, after several years on Autoimmunity Research Foundation’s Marshall Protocol, this grandfather has largely recovered from each of his diseases, to the point where he is barely conscious of symptoms and has extra time for work and play. Meet Chris Eastlund.

Can you describe the progression of your symptoms?

Things first started to go wrong when I was around 8-10 years old. I spent an entire summer just sleeping on my grandmother’s couch. I’m told I was only awake for about four hours a day. I now realize that such fatigue is one of the symptoms of pediatric Lyme, but when I was taken to the Mayo Clinic I was told nothing was wrong with me.

I pushed on, but could never play endurance sports. I could never run for even 1/4 of a mile before having to stop. At first I thought I could join the cross country team and train my body to handle more exercise. While running I would feel better, but then a few hours later, I would suffer from feelings of fatigue and soreness - what is often referred to as post-exertional malaise.

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In 2005, this father of seven could hardly breathe and suffered from intense joint pain. Exhausted and sore, he found it extremely difficult to walk up the stairs. Now, after about 2 1/2 years on Autoimmunity Research Foundation’s Marshall Protocol he’s essentially pain and symptom free and is back to digging trenches in his garden. Roy P. will now take your questions.

Can you describe the progression of your disease?

Everything started around Christmas of 2000. I was at work when my legs started to swell down in the area by my ankles. I was also in pain, couldn’t walk, and had a very difficult time breathing. A co-worker called my wife who took me home. We saw a doctor and I was diagnosed with rheumatoid arthritis. However the doctor still wasn’t able to explain why my legs were swollen, or why I was suffering from other severe symptoms. Personally, I thought I was having a heart attack. At that point I asked, “Do you think any of this is related to the lumps I have under the bicep of my right arm?” He paused, and said, “What lumps? Show me.” After examining my arm he said “Wait a minute, I know what else you might have….sarcoidosis!” I proceeded to have a series of lung X-rays done which confirmed that I did indeed have the disease.

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As humans, our environment consistently exposes us to a variety of dangers. Tornadoes, lightning, flooding and hurricanes can all hamper our survival. Not to mention the fact that most of us can encounter swerving cars or ill-intentioned people at any given moment.

Biofilms form when bacteria adhere to surfaces in aqueous environments and begin to excrete a slimy, glue-like substance that can anchor them to all kinds of material

Thousands of years ago, humans realized that they could better survive a dangerous world if they formed into communities, particularly communities consisting of people with different talents. They realized that a community is far more likely to survive through division of labor– one person makes food, another gathers resources, still another protects the community against invaders. Working together in this manner requires communication and cooperation.

Inhabitants of a community live in close proximity and create various forms of shelter in order to protect themselves from external threats. We build houses that protect our families and larger buildings that protect the entire community. Grouping together inside places of shelter is a logical way to enhance survival.

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Four years ago this Australian native’s joint and muscle pain was so bad that he was barely able to walk, and his cognitive function had diminished to the point where he could hardly think straight. Plagued by intense chemical sensitivities, his days were spent indoors wearing a carbon respirator. Today, after about three years on Autoimmunity Research Foundation’s Marshall Protocol, he could talk for hours about how much better he feels and his wide array of symptoms are essentially gone. Meet Peter de Jager.

Can you describe the progression of your disease?

It’s difficult to tell when I first started to feel sick, since it developed so slowly. Even as a teenager I wondered how people could run long distances, breathing through their nose! – I couldn’t, I was already a mouth-breather.

In my early twenties I had a bout of glandular fever (Epstein-Barr virus) from which I took a long time to recover. In the late 1970s I constantly had small scabby sores in my nose.

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In the following video, I explain the science that forms the backbone of the Marshall Protocol in simple terms. I discuss the bacteria implicated in causing chronic inflammatory disease, and explain how these pathogens are hypothesized to affect the immune system. I also describe the basics of the treatment itself, providing information that any patient who plans to start the Marshall Protocol, or any doctor planning to put a patient on the MP, should understand. My narration is accompanied by slides with pictures and images of the pathogens and molecules I discuss. The video is 85 minutes.

Please note– clicking on the “x” shape in the bottom right corner of the video, just to the left of the word “vimeo” will give you a full-screen version of the presentation. (Nice, right?)

Want to give some or all of this presentation to researchers, doctors, or patients? Be my guest! I should say this presentation was made in a program called Keynote. The Keynote presentation is available upon request. I’ve made the PDF version of this presentation as well.

At the 2008 Days of Molecular Modeling Conference in Sweden, biomedical researcher Trevor Marshall sat on the edge of his chair listening intently to a talk presented by Adriano Aguzzi of the University Hospital of Zurich. Aguzzi was discussing research that confirmed much of what Marshall had long suspected to be true about prions - small, potentially infectious molecules that are hypothesized to be made only of protein.

The protein structure of a prion.

Prions have been implicated as the cause of a number of diseases in a variety of mammals, including bovine spongiform encephalopathy (BSE, also known as “mad cow disease”) in cattle, and Creutzfeldt-Jakob disease (CJD) in humans. All thus-far hypothesized prion diseases affect the structure of the brain or other neural tissue, and all are considered untreatable or fatal by mainstream medicine.

Although prions have been studied to some extent in the lab for decades, very little research has delved into their actions when inside the human body (in vivo). Thus, many theories put forth about how prions might cause or contribute to neurological disease have been largely speculative.

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